How to identify corn borer damage

Corn borer, also known as corn borer, is a worldwide corn pest. Corn borer is a polyphagous pest with up to 200 host plants, but the main crops are corn, sorghum, and millet.
1. Symptom identification: Corn borer larvae are borers, and the typical symptom is that after the heart leaves are pierced, the unfolded corn leaves appear neat rows of small holes. After the tassels are withdrawn, the corn borer larvae injure male flowers, often causing the male flower base to break. After the emergence of this ear, the larvae transfer the female ear to take the filaments and tender tenderloin leaves, and enter the cob or eat the young seeds. Another part of the larvae from the stem and leaf sheath into the stem, feeding pith, so that the stem easily blown by the wind. The grains of the affected plants are not full, with premature aging of blue-green, and some spikes even without grains, resulting in serious reduction of yield.
The life of corn borer is also divided into four stages: adult (moth), egg, larvae, and pupa. The adult corn borer is smaller than the ground tiger. Female adult cephalothorax is light brown on the back, ventral and foot white, yellowish-brown forebrain, with 3 dark wavy lines; abdomen and hind wings yellow-white, with dark curved lines in the middle and proximal part of the hind wings. The head of the male head is milky white, with reddish-brown or dark yellow-brown wings; the hind wings are light brown with 2 bands. On the surface of eggs, there are polygonal reticular patterns of different sizes, which are milky at the time of first production, and turn pale yellow or light green afterwards. They are dark brown before hatching, and they produce irregularly shaped egg masses, each of which has 15-60 eggs. Scale arrangement of fish. The larvae are white, with a pink, gray or taupe on the back, head

Vitamins & Nutritions

Vitamins:

Vitamin A: Retinol. Carotene compounds responsible for transmitting light sensation in the retina of the eye. Deficiency leads to night blindness.

Beta carotene: An antioxidant which protects cells against oxidation damage that can lead to cancer. Beta carotene is converted, as needed, to vitamin A. Food sources of beta carotene include vegetables such as carrots, sweet potatoes, spinach and other leafy green vegetables; and fruit such as cantaloupes and apricots. Excessive carotene in the diet can temporarily yellow the skin, a condition called carotenemia, commonly seen in infants fed largely mushed carrots.

Vitamin B1: Thiamin, acts as a coenzyme in body metabolism. Deficiency leads to beriberi, a disease of the heart and nervous system.

Vitamin B2: Riboflavin, essential for the reactions of coenzymes. Deficiency causes inflammation of the lining of the mouth and skin.

Vitamin B3: Niacin, an essential part of coenzymes of body metabolism. Deficiency causes inflammation of the skin, vagina, rectum and mouth, as well as mental slowing.

Vitamin B6: Pyridoxine, a cofactor for enzymes. Deficiency leads to inflammation of the skin and mouth, nausea, vomiting, dizziness , weakness and anemia.

Folate (folic acid): Folic acid is an important factor in nucleic acid synthesis (the genetic material). Folate deficiency leads to megaloblastic anemia.

Vitamin B12: An essential factor in nucleic acid synthesis (the genetic material of all cells). Deficiency leads to megaloblastic anemia, as can be seen in pernicious anemia.

Vitamin C: Ascorbic acid, important in the synthesis of collagen, the framework protein for tissues of the body. Deficiency leads to scurvy, characterized by fragile capillaries, poor wound healing, and bone deformity in children.

Vitamin D: A steroid vitamin which promotes absorption and metabolism of calcium and phosphorus. Under normal conditions of sunlight exposure, no dietary supplementation is necessary because sunlight promotes adequate vitamin D synthesis in the skin. Deficiency can lead to osteomalacia in adults and bone deformity (rickets) in children.

Vitamin E: Deficiency can lead to anemia.

Vitamin K: An essential factor in the formation of blood clotting factors. Deficiency can lead to abnormal bleeding.


Nutritions:

For the treatment of nutritional disease, any of the nutrient-related diseases and conditions that cause illness in humans. They may include deficiencies or excesses in the diet, obesity and eating disorders, and chronic diseases such as cardiovascular disease, hypertension, cancer, and diabetes mellitus. Nutritional diseases also include developmental abnormalities that can be prevented by diet, hereditary metabolic disorders that respond to dietary treatment, the interaction of foods and nutrients with drugs, food allergies and intolerances, and potential hazards in the food supply. All of these categories are described in this article. For a discussion of essential nutrients, dietary recommendations, and human nutritional needs and concerns throughout the life cycle, see nutrition, human.

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